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    Revista de Actualización Clínica Investiga

    versão impressa ISSN 2304-3768

    Resumo

    VILLALBA HERRERA, Ericka Wendie. INFLAMACION I. Rev. Act. Clin. Med [online]. 2014, vol.43, pp. 2261-2265. ISSN 2304-3768.

    Inflammation is a natural physiological process of the body's defense against environmental assaults, presenting signs such as pain, heat, redness and edema, in addition to loss of functionality. This process can occur acutely with immediate reaction to the stressor, in which phagocytes, upon encountering an offending agent secreting mediators try to destroy substances that act on endothelial cells causing changes in vascular permeability, allowing the migration of leukocytes the inflammatory focus for phagocytosis of pathogens, returning to normal when the injury was restored. Likewise, the inflammation progresses to persistent infections, as a result of evolution of autoimmunity; or a continuation of acute inflammation as the injurious agent persists, leading to chronic inflammation. At this level of mononuclear cell infiltration, tissue destruction and attempts at healing of damaged tissue by angiogenesis or fibrosis occurs. The purpose of inflammation is to rid the body of the initial cause of cell aggression, however whatever the inflammatory acute or chronic condition, you should take appropriate measures to control the sequelae of inflammation in the body without interfering with pharmacological measures its beneficial effects.

    Palavras-chave : Inflamation; Edema; Exudate; Trasudate; Stasis; Suffusión; Hydrostatic pressure; Osmotic pressure; Chemotaxis; Fagocitosis.

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